Formula Inhibits Tumor Growth and Genes Related to Metastasis Results of the study display that the oral administration of the formula produced a statistically significant suppression of tumor growth, compared to controls. The study was performed utilizing a xenograft model of human prostate malignancy in mice. A xenograft model is usually when cells or organs from an individual of 1 species are transplanted or grafted onto an organism of another species, genus, or family. So individual prostate cancer cells are grafted onto a laboratory animal, which in cases like this had been mice. Related StoriesCrucial change in single DNA foundation predisposes children to intense form of cancerNew results reveal association between colorectal cancer and melanoma drug treatmentNew RNA check of blood platelets can be used to identify location of cancerIn addition to a substantial decrease in tumor volume, results of this study showed the method also worked well to inhibit a number of genes involved in malignancy proliferation and metastasis.AWMSG guidance. Data on file. New England Journal of Medication 2011; 364:11-21. British Heart Base Health Promotion Analysis Group, Department of Community Health. Coronary heart disease figures. University of Oxford. 2010 edition. Stewart S, et al. More 'malignant' than malignancy? Five-year survival following a first admission for heart failing. European Journal of Cardiovascular Failing. 2001 Jun;3:315-22. The cardiac disease National Service Framework for Wales. 2009. McMurray J, et al. The pharmacoeconomics of ACE inhibitors in persistent heart failure. Pharmacoeconomics 1996;9:188-197. Stewart S, et al. The existing cost of heart failure to the National Wellness Service in the united kingdom.